Where is the study no1?

@metal-brain said
They are invalid studies and I proved that. Remember the article by Hatfill? He is the same person Mueller went on a witch hunt attempting to falsely convict him of the anthrax attack. You have bit off more than you can chew.

The link you provided even included a Chloroquine study. That is NOT Hydroxychloroquine! But hey, they were desperate and needed to include a dif ...[text shortened]... on it. Post a study one by one and I will debunk them one by one. They are all designed to mislead.

@athousandyoung said

no1 - I do recall its effectiveness as a treatment for COVID 19 was questioned

That's what your links are saying sh76 - COVID plus HQ leads to increased death, not HQ alone.

Last Spring, the link between Hydroxychloroquine [used in COVID patients] and heart-related deaths was all over outlets like WaPo and CNN.

Much better.

@sh76 said
That is a distinction without a difference.

The entire context of the discussion was about HQ in the context of treating COVID; not HQ in a vacuum.

All Spring there were reports about HQ as a treatment for COVID being more dangerous than no treatment at all. Those reports are what turned out to be unsubstantiated and that is the whole point of this discussion. Obviously, whether HQ is dangerous outside the context of COVID is completely irrelevant here.

@athousandyoung said
Not at all because the defenders of HQ kept blabbing about how we know it is safe because it's used for malaria

@sh76 said
Yes; and that turned out to be an excellent point; didn't it?

From Feb 2021:

https://pubmed.ncbi.nlm.nih.gov/33163895/

Conclusion: Treatment of patients with COVID-19 with CQ or HCQ is associated with an important risk of drug-induced QT prolongation and relatively higher incidence of torsades de pointes, ventricular tachycardia, or cardiac arrest. Therefore, these agents should not be used routinely in the management of COVID-19 disease. Patients with COVID-19 who are treated with antimalarials for other indications should be adequately monitored.

@athousandyoung said
From Feb 2021:

https://pubmed.ncbi.nlm.nih.gov/33163895/

Conclusion: Treatment of patients with COVID-19 with CQ or HCQ is associated with an important risk of drug-induced QT prolongation and relatively higher incidence of torsades de pointes, ventricular tachycardia, or cardiac arrest. Therefore, these agents should not be used routinely in the managemen ...[text shortened]... -19 who are treated with antimalarials for other indications should be adequately monitored.

@sh76 said
There are many possible COVID treatments. Ivermectin has a better record than Hydroxychloroquine, but weaker than Tocilizumab and Fluvoxemine. I doubt the latter two cost much either.

Why would I give a damn about profits for Big Pharma?

@metal-brain said
Again, what is your source of information?
Do you accept my assertions without proof? Of course not, so why should I accept your assertions after failing to show me your source twice?

@metal-brain said
"Objective: To systematically review the literature and to estimate the risk of chloroquine (CQ) and hydroxychloroquine (HCQ) cardiac toxicity in patients with coronavirus disease 2019 (COVID-19)."

From the link below:

"Nowhere was there any mention of the fact that COVID-19 damages the heart during infection, sometimes causing irregular and sometimes fatal heart r ...[text shortened]... (CQ) and hydroxychloroquine (HCQ) together. This is about HCQ, not CQ. They are not the same thing.

https://www.ahajournals.org/doi/10.1161/JAHA.120.016887

Chloroquine and hydroxychloroquine were recognized as impacting the electrophysiological properties of the heart decades ago. Clinically, they can prolong the QT interval that could potentially initiate ventricular arrhythmias including torsades de pointes. Chloroquine is known to be an open channel blocker of the hERG 1A and 1A/1B potassium channel that in the heart underlies the repolarizing potassium current IKr; chloroquine binds to the common drug‐binding site in the channel pore. Block of IKr is the principal cause of drug‐induced long QT syndrome. Chloroquine also binds to cardiac sodium, calcium, and inward rectifier potassium channels to potentially cause QRS widening and conduction abnormalities.

@athousandyoung said
Your source of information is sensationalist, partisan journalism. Show me the peer reviewed science like I showed you. Pubmed is free!

https://pubmed.ncbi.nlm.nih.gov/

https://www.ahajournals.org/doi/10.1161/JAHA.120.016887

[b]Chloroquine and hydroxychloroquine were recognized as impacting the electrophysiological properties of the heart decades ago[ ...[text shortened]... rectifier potassium channels to potentially cause QRS widening and conduction abnormalities.

@metal-brain said
Again, what is your source of information?
Do you accept my assertions without proof? Of course not, so why should I accept your assertions after failing to show me your source twice?

@athousandyoung said
No, it was irrelevant to the claim that HQ and COVID led to heart problems

@sh76 said
Let's start with the RECOVERY trial results.

https://www.recoverytrial.net/results

Have you all watched the news lately? They are investing money into finding a pill to treat C19....lol! We already have HCQ and ivermectin that are proven to be effective and safe.

They want a drug that is under patent protection so they can charge an arm and a leg for it. Isn't it obvious? That is why they lie about low cost drugs and create flawed studies with fraudulent data. They think you are all stupid!